CVR Physiotherapy

CVR Physiotherapy › Chapter 6 › 6(h) and 6(i) Diabetes

Diabetes, diabetic foot, wound healing and diabetes insipidus

Diabetes is part of many long cases (CABG, MI, PAD, amputation, varicose veins) and was asked on its own as 7-mark questions on glycaemic control and diabetic foot care. Newer papers ask about wound healing and pressure sores. Learn the exercise prescription, the precautions, foot care with its reasons, and wound care.

Syllabus
Ch 6(i) Metabolic syndrome: diabetes mellitus and insipidus; 6(h) diabetic ulcers
Priority
Tier 2. 7-mark questions on diabetes (S16, W18, S19) and on wounds (W19 pressure sore; S24 and S25 wound healing); a co-existing condition in 10 long cases.
Asked as
7m S16W18S19W19S24S25 3m S16S19S22
Usual split
7-mark answers; part of long answers on CABG, PAD, amputation and venous disease
Checked against
ADA Standards of Care (2026); ADA position statement on physical activity and diabetes (2016); IWGDF diabetic foot guidelines (2023); 2022 renaming of diabetes insipidus. Full list under Sources.
Content checked
19 October 2026. Next review due January 2027 (after ADA 2027).
My revision

1Definition

Diabetes mellitus is a group of metabolic disorders with a raised blood glucose, caused by lack of insulin, poor response to insulin, or both.

Diabetic foot is infection, ulceration or destruction of tissues of the foot in a person with diabetes, usually because of nerve damage (neuropathy) and/or peripheral arterial disease.

Diabetes insipidus (now called arginine vasopressin (AVP) deficiency or AVP resistance) is a different disease: the body cannot concentrate urine, so the person passes large volumes of dilute urine and is always thirsty. Blood sugar is normal.

2Classification and grading

Types of diabetes mellitus

  • Type 1: autoimmune destruction of insulin-producing cells; usually young; needs insulin; risk of ketoacidosis.
  • Type 2: insulin resistance with failing insulin secretion; linked to obesity, inactivity and family history; the commonest type, and common in younger Indians.
  • Gestational diabetes in pregnancy; other specific types (pancreatic disease, steroids).

Diagnosis (ADA)

TestDiabetesPrediabetes
Fasting plasma glucose126 mg/dL or more100–125 mg/dL
2-hour glucose after 75 g glucose drink200 mg/dL or more140–199 mg/dL
HbA1c6.5% or more5.7–6.4%
Random glucose with symptoms200 mg/dL or more–

Diabetic foot ulcer: Wagner grades

GradeDescription
0At-risk foot, no open wound (callus, deformity)
1Superficial ulcer
2Deep ulcer reaching tendon, capsule or bone, no abscess
3Deep ulcer with abscess or osteomyelitis
4Gangrene of part of the foot (toes or forefoot)
5Gangrene of the whole foot

Foot risk categories (IWGDF)

  • 0, very low: no loss of protective sensation and no PAD. Screen yearly.
  • 1, low: loss of protective sensation or PAD. Screen every 6–12 months.
  • 2, moderate: two of: loss of sensation, PAD, foot deformity. Screen every 3–6 months.
  • 3, high: loss of sensation or PAD plus a past ulcer, past amputation or end-stage kidney disease. Screen every 1–3 months.

3Causes and risk factors

  • Type 2: obesity, especially abdominal; physical inactivity; family history; South Asian ethnicity (diabetes at lower BMI and younger age); unhealthy diet; age; gestational diabetes in the past.
  • Diabetic foot: long duration, poor sugar control, neuropathy, PAD, foot deformity, smoking, kidney disease, walking barefoot, poor footwear, previous ulcer.

4Pathophysiology to impairments

Why the diabetic foot ulcerates: three causes

  • Sensory neuropathy: loss of protective feeling, so injuries, tight shoes and burns are not noticed.
  • Motor neuropathy: weak small foot muscles cause claw toes and prominent metatarsal heads, so pressure under the forefoot rises and callus forms.
  • Autonomic neuropathy: no sweating, so dry, cracked skin lets bacteria in.
  • Ischaemia (PAD) slows healing; high sugar weakens immunity, so infection spreads fast.

Repeated pressure under a callus causes bleeding and breakdown beneath it: a neuropathic ulcer. Add infection and ischaemia, and the result can be gangrene and amputation.

Other complications that affect physiotherapy

  • Heart and vessels: heart attack (often silent), stroke, PAD.
  • Eyes: retinopathy (bleeding risk with straining and head-down positions).
  • Kidneys: nephropathy.
  • Autonomic neuropathy: postural hypotension, fixed heart rate, poor heat tolerance.
  • Musculoskeletal: frozen shoulder, stiff hands, Charcot foot (collapse of foot joints).

Stages of wound healing (3 marks in S19)

Haemostasis (minutes to hours) → inflammation (about days 1–5) → proliferation (about day 4 to week 3) → remodelling (week 3 to 1–2 years). See the full table on the burns page. Diabetes slows every stage, especially inflammation and proliferation.

Factors affecting wound healing (7 marks in S25)

  • Local: infection; poor blood supply (arterial disease, pressure, tight dressings); oedema; dead tissue or foreign bodies; repeated shear, friction or pressure; a wound that is too dry or too wet; a large or deep wound; previous radiotherapy.
  • General: older age; poor nutrition (protein, vitamin C, zinc); diabetes with high sugar; smoking; anaemia and low oxygen; obesity; drugs (steroids, chemotherapy); kidney or liver failure; reduced immunity; stress and poor sleep.

In a diabetic foot the main ones are high sugar, ischaemia, unnoticed pressure from neuropathy, infection and smoking. Answer S25 with this list plus the modalities in section 12.

Recall check. Write the three neuropathy types and how each leads to a foot ulcer.

5Clinical features

  • Diabetes: thirst, frequent urination, tiredness, weight loss, blurred vision, slow-healing wounds, repeated infections; many type 2 patients have no symptoms.
  • Neuropathic foot: numbness, tingling or burning, warm dry skin, callus, claw toes, good pulses (unless PAD), painless ulcer under the forefoot or heel.
  • Ischaemic foot: cold, pale, absent pulses, painful ulcers at toes and heel.
  • Diabetes insipidus: passing large amounts of pale urine (more than about 3 litres a day in adults), waking at night to pass urine, strong thirst, dehydration if water is not available.
Hypoglycaemia (blood sugar below 70 mg/dL)

Sweating, shaking, hunger, fast heart rate, confusion, slurred speech, unusual behaviour, seizures. Can happen during or many hours after exercise, especially with insulin or sulfonylureas.

6Investigations and foot tests

TestWhat it showsPhysio meaning
HbA1cAverage sugar over about 3 monthsPoor control means slow healing, more care with exercise
Glucometer readingSugar nowCheck before and after exercise if on insulin or sulfonylureas
10 g monofilamentLoss of protective sensationFoot risk category; protective footwear
128 Hz tuning fork, ankle reflexesLarge-fibre neuropathyBalance and fall risk
Pulses, ABI, toe pressuresPAD (ABI may be falsely high from calcified arteries)Ischaemic foot needs vascular review
Lipids, kidney function, ECG, eye checkComplicationsModify exercise for heart, eye and kidney disease
Water deprivation test, sodiumDiabetes insipidusAllow free access to water during sessions

7Medical management

  • Lifestyle: diet, physical activity, weight loss, stopping smoking.
  • Medicines: metformin; sulfonylureas (cause hypoglycaemia); DPP-4 inhibitors; SGLT2 inhibitors and GLP-1 receptor agonists (also protect the heart and kidneys); insulin.
  • Usual target HbA1c around 7% for many adults, set individually.
  • Control BP and lipids; yearly eye, kidney and foot checks.
  • Diabetic foot ulcer: off-loading (for example total contact cast), debridement, infection control, revascularisation if ischaemic, good sugar control.
  • Diabetes insipidus: treat the cause; desmopressin for AVP deficiency.

8Complications and prevention

ComplicationWhat to look forPhysio role
HypoglycaemiaSweating, shaking, confusion during or after exerciseCheck sugar; keep glucose tablets ready; rule of 15
Foot ulcer, infection, amputationCallus, blisters, redness, warmth, smellFoot checks every session, footwear advice, off-loading
Silent heart attackBreathlessness or fatigue without chest painMonitor HR and BP; use RPE
Retinal bleedingSudden blurred visionAvoid heavy straining, breath-holding, jumping and head-down positions in advanced retinopathy
FallsNumb feet, poor balance, postural hypotensionBalance training, safe footwear, slow position changes
Frozen shoulder, stiff handsPainful restricted shoulderROM exercises, mobilisation

9PT assessment

  • Type and duration of diabetes, medicines (insulin, sulfonylureas), recent HbA1c and readings, hypoglycaemia episodes, complications.
  • Activity level, occupation, diet habits, smoking, weight, BMI and waist.
  • Feet: skin, nails, callus, deformity, footwear, monofilament sensation, vibration, pulses, ulcer description (site, size, depth, Wagner grade).
  • BP lying and standing; resting HR; fitness (6MWT); strength; balance (single-leg stance, Timed Up and Go).
  • Outcome measures: HbA1c, weight and waist, 6MWT, ulcer area, balance tests, quality of life.

10PT diagnosis by ICF

Diabetes appears inside other long cases. This practice case is built from those (not a single past paper).

Practice case: a 55-year-old shopkeeper with type 2 diabetes for 12 years, HbA1c 9%, numb feet, callus under the first metatarsal head, sedentary, overweight. He walks barefoot at home and in the shop.

ICF componentFrom this caseClinical reasoning
Health conditionPoorly controlled type 2 diabetes with peripheral neuropathy; high-risk footLong duration and high HbA1c
Body function impairmentsLoss of protective sensation; callus and raised forefoot pressure; reduced fitness; excess weight; reduced balanceNeuropathy plus inactivity
Activity limitationsSafe walking on uneven ground; long standing in the shopNumb feet and callus pain
Participation restrictionsAt risk of losing work days to ulcers; limited social walkingUlcer or amputation would stop work
Environmental factorsBarriers: barefoot habit, sweets and fried snacks at the shop. Facilitators: family, local clinic.Education targets habits
Personal factors55-year-old man, low awareness of foot riskTeach "why", not just "what"

11Problem list and goals

  • Short-term (4 weeks): daily foot inspection habit; protective footwear always; walking 20 minutes on most days with safe sugar readings; knows hypoglycaemia signs and treatment.
  • Long-term (3–6 months): 150 minutes a week of aerobic activity plus resistance training twice a week; HbA1c and weight reduced; no ulcers; better balance.

12PT management

Exercise for glycaemic control (7 marks in S16 and W18)

Why exercise works: contracting muscle takes up glucose even without insulin; after training, insulin sensitivity improves for a day or more; exercise also reduces weight, BP, lipids and heart risk.

ADA prescription for adults with type 2 diabetes
TypeDose
AerobicAt least 150 min a week of moderate-to-vigorous activity, spread over 3–7 days, no more than 2 days in a row without exercise
Resistance2–3 sessions a week on non-consecutive days; 1–3 sets of 10–15 repetitions to near fatigue at first
Flexibility and balance2–3 days a week; stretches held 10–30 s, 2–4 repetitions; balance work for older adults and those with neuropathy
Less sittingA few minutes of light activity every 30 minutes of sitting
Memory hook: hypoglycaemia "rule of 15"

15 g, then 15 minutes

If sugar is below 70 mg/dL or symptoms appear: give 15 g of fast sugar (glucose tablets, 3 teaspoons of sugar in water, half a glass of juice), recheck after 15 minutes, repeat if still low, then a snack.

Diabetic foot care (7 marks in S19: "what is diabetic foot, how to take care of it, and why")

CareHowWhy
Inspect dailyLook at soles and between toes with a mirror or a family memberNumb feet do not feel injuries
Wash and dryLukewarm water (test with the elbow), dry between toesPrevents burns and fungal infection
MoisturiseCream on dry skin, not between the toesDry skin cracks and lets bacteria in
Nail careCut straight across; no home surgery on cornsPrevents ingrown nails and wounds
FootwearSoft, roomy, closed shoes with socks; check inside before wearing; never barefoot (home, temple, beach)Protects against unnoticed injury and pressure
Avoid heatNo hot-water bottles, heating pads or walking on hot surfacesBurns are not felt
Control sugar, stop smokingMedicines, diet, exerciseBetter healing and circulation
Seek help earlyAny blister, cut, redness or swellingSmall wounds become ulcers in days
  • With an active plantar ulcer: off-load the foot (cast, boot, crutches); switch to non-weight-bearing exercise such as cycling with the other leg, arm ergometry or swimming once the wound allows.
  • Neuropathy without ulcer: moderate weight-bearing walking is safe with proper footwear and daily checks.

Electrotherapy for wound healing (3 marks in S16 and S22; 7 marks in S24 and S25)

Typical textbook parameters (vary between sources; follow local protocol)
ModalityTypical parametersEffect
High-voltage pulsed current (electrical stimulation)Twin-peak monophasic pulses, about 100 pulses per second, sensory-level voltage (often 100–150 V), 45–60 minutes, daily or 5 days a week; negative pole on an infected wound, positive to promote healingAttracts repair cells, improves blood flow, antibacterial effect
Therapeutic ultrasoundPulsed 20%, about 0.5 W/cm², 3 MHz, 5–10 minutes on the skin around the wound (not into an open wound without a sterile coupling)Stimulates inflammation to move on, collagen formation
Ultraviolet (UVC)Short-wave UV (about 254 nm), short exposures by test dose, protecting surrounding skin and eyesKills bacteria in infected wounds
Low-level laserLow dose, about 1–4 J/cm² to wound edgesCell stimulation, healing

Exact parameters differ between electrotherapy books and trials. Write a range with the effect, and never use heat or electrotherapy over an ischaemic or numb area without checking circulation.

Pressure sores (pressure injuries) (7 marks in W19)

Damage to skin and the tissue under it from pressure, or pressure with shear, usually over a bony point: sacrum, heels, hips (greater trochanter), ischial tuberosities, back of the head, elbows. Patients at risk: bed- or chair-bound, numb skin (diabetes, spinal cord injury), ICU patients, poor nutrition, incontinence, older age.

Stages (NPIAP, 2016)
StageWhat you see
Stage 1Intact skin with redness that does not fade (blanch) when pressed
Stage 2Partial-thickness skin loss: shallow pink wound or blister; fat not visible
Stage 3Full-thickness skin loss: fat visible; no bone, tendon or muscle seen
Stage 4Full-thickness skin and tissue loss: muscle, tendon, ligament, cartilage or bone exposed or felt
UnstageableBase covered by slough or eschar, so depth cannot be seen
Deep tissue pressure injuryIntact or broken skin with a persistent deep red, maroon or purple area

Risk assessment: Braden scale (sensory perception, moisture, activity, mobility, nutrition, friction and shear; total 6–23). A score of 18 or less means the patient is at risk; the lower the score, the higher the risk.

Physiotherapy

  • Relieve pressure: reposition on a schedule set for that patient; 30° tilted side-lying rather than lying straight on the hip; head of bed no higher than 30° unless breathing needs more; heels floated off the bed with a pillow under the calves.
  • Surfaces: high-specification foam or alternating-pressure air mattress; pressure-relieving cushion in the chair; teach chair users to shift weight (push-ups or leaning) regularly.
  • Move: bed mobility, sitting out and walking as early as possible; active and passive exercise to keep circulation and joint range.
  • Avoid shear: lift, do not drag; use slide sheets.
  • Help the wound heal: electrical stimulation has the best evidence of the physical modalities for stage 2 sores that are slow to heal and for stage 3–4 sores (parameters as in the table above); wound cleaning and dressing by the team; nutrition and continence care.
  • Teach: daily skin checks, weight shifts, never sit or lie on a red area.

Classic answer vs current guideline: turning

Classic

Turn every patient every 2 hours.

Current (international guideline, 2019)

Set the turning frequency for each patient from their risk, skin checks, comfort and the mattress they are on.

Write in the exam

"Reposition at least every 2 hours, or as the patient's skin and support surface require."

Diabetes insipidus

  • Allow free access to water and toilets during sessions; watch for dehydration (dizziness, dry mouth, fast pulse).
  • Night-time urination raises the fall risk in older patients: night light, clear path.
  • Seen in ICU after head injury or pituitary surgery: monitor fluid balance before mobilising.
Recall check. Write the ADA exercise prescription, five foot-care rules with their reasons, and the six pressure-sore stages.

13Precautions

Exercise precautions in diabetes
  • Check sugar before and after exercise if on insulin or sulfonylureas; carry fast sugar; avoid exercising at the peak action of insulin.
  • Treat a low sugar first and do not start until it is back to normal.
  • Avoid exercise when sugar is very high with ketones (mainly type 1).
  • Advanced retinopathy: avoid vigorous exercise, heavy lifting, jumping, head-down positions and breath-holding.
  • Neuropathy: proper footwear; check feet after each session; balance training.
  • Autonomic neuropathy: change position slowly; use RPE instead of heart rate; avoid hot, humid days; drink water.
  • Heart disease is often silent: report unusual breathlessness or fatigue.
  • No heat or ice on numb skin; check circulation before any electrotherapy.

14Education and lifestyle

  • Be active every day; break up sitting every 30 minutes.
  • Eat regular meals with fewer refined carbohydrates and sweets; more vegetables, pulses and fibre.
  • Reach a healthy weight and waist (Indian targets: BMI below 23; waist below 90 cm in men, 80 cm in women).
  • Take medicines regularly; know your hypoglycaemia plan.
  • Daily foot care for life; yearly eye, kidney and foot checks.
  • Stop smoking and tobacco.

15Prognosis

Good control of sugar, BP and lipids, regular exercise and daily foot care greatly reduce complications. Most diabetic amputations start with a foot ulcer that could have been prevented.

16Exam corner

Past questions (MUHS, 2016–2026)

  • 7-mark questionsPhysiotherapy for glycaemic control in diabetes (S16). Role of physiotherapy in diabetes (W18). What is diabetic foot, how to care for it, and why (S19). Physiotherapy for pressure sores (W19). Modalities for wound healing (S24). Factors affecting wound healing, with the modalities used (S25).
  • 3-mark questionsThree electrotherapy modalities for an infected wound (S16). Stages of wound healing (S19). Three electrotherapy modalities with parameters for wound healing (S22).
  • Inside long casesDiabetes with CABG (W18, S21), PAD (W22), amputation (S18, S19), varicose veins (S17), angina with EF 25% (S20), amputation (W19, S23, S25), MI (S26).

Technique cards for this topic: exercise physiology, exercise testing, electrotherapy and exercise.

Keywords to underline

insulin sensitivity 150 min a week no more than 2 days without exercise resistance 2–3 times a week hypoglycaemia rule of 15 loss of protective sensation monofilament off-loading daily foot inspection

Viva questions

  1. How does exercise lower blood sugar?
  2. What would you do if a patient becomes sweaty and confused during exercise?
  3. Why do neuropathic ulcers form under the metatarsal heads?
  4. Which exercises should a patient with proliferative retinopathy avoid?
  5. How is diabetes insipidus different from diabetes mellitus?

Test yourself

Answer on paper first, then open the answer.

7 mPhysiotherapy management for glycaemic control in diabetes mellitus.

Why exercise works; assessment and screening; aerobic and resistance prescription (ADA doses); flexibility and balance; breaking up sitting; precautions (hypoglycaemia, retinopathy, neuropathy, autonomic); foot care; education; monitoring outcomes (HbA1c, weight).

3 mThree electrotherapeutic modalities with parameters for wound healing.
  1. High-voltage pulsed current: about 100 pps, sensory level, 45–60 min, negative pole for infected wound.
  2. Pulsed ultrasound: 20%, about 0.5 W/cm², 3 MHz, 5–10 min around the wound.
  3. UVC about 254 nm, short test-dosed exposure for bacterial kill. (Laser 1–4 J/cm² also accepted.)
MCQA patient on insulin feels shaky and sweaty mid-session; glucometer shows 60 mg/dL. Best action:A. Continue at lower intensityB. Stop, give 15 g fast sugar, recheck in 15 minutesC. Give insulinD. Tell him to rest at home

B. The rule of 15 treats mild hypoglycaemia safely.

MCQWhich footwear advice is correct for a diabetic with numb feet?A. Walk barefoot at home to strengthen the feetB. Tight shoes for supportC. Soft, roomy closed shoes with socks, checked inside before wearingD. Hot-water soaks every night

C. Numb feet cannot sense injury; protection and daily checks prevent ulcers.

 Sources

Notes written in original words. Numbers were checked against the sources below.

  1. MUHS BPTh syllabus (2012) and MUHS IV BPTh question papers 2016–2026 (muhsonline.com, unofficial archive; Dhole Patil College of Physiotherapy past-paper page).
  2. American Diabetes Association. Standards of Care in Diabetes 2026. Diabetes Care 2026.
  3. Colberg SR, et al. Physical Activity/Exercise and Diabetes: A Position Statement of the American Diabetes Association. Diabetes Care 2016;39:2065–79. Key points summary: PMC.
  4. International Working Group on the Diabetic Foot (IWGDF) Guidelines 2023.
  5. Arima H, et al. Changing the name of diabetes insipidus: a position statement. 2022.
  6. Electrical stimulation parameters: reviews of high-voltage pulsed current in chronic wounds (PMC); electrotherapy.org.
  7. RSSDI clinical practice recommendations for type 2 diabetes (India), 2022.
  8. National Pressure Injury Advisory Panel. Pressure injury stages (2016). npiap.com
  9. European Pressure Ulcer Advisory Panel, National Pressure Injury Advisory Panel and Pan Pacific Pressure Injury Alliance. Prevention and treatment of pressure ulcers/injuries: clinical practice guideline, 3rd edition (2019).

Educational notes for exam preparation. Not medical advice. Hospital protocols and the treating doctor's instructions come first.

Version 1. Content checked 19 October 2026. Next review January 2027.